(2007)
| Titre : |
Addressing challenges in adolescent smoking cessation : design and baseline characteristics of the HS Group-Randomized trial |
| Type de document : |
texte imprimé |
| Auteurs : |
Jingmin Liu, Auteur ; Jr. Peterson, Auteur ; V. Arthur, Auteur ; Kathleen A. Kealey, Auteur ; Sue L. Mann, Auteur ; Jonathan B. Bricker, Auteur ; Patrick M. Marek, Auteur |
| Editeur : |
Paris [France] : Elsevier |
| Année de publication : |
2007 |
| Collection : |
Preventive Medecine |
| Note générale : |
Dans la bibliothèque virtuelle (articles scientifiques) |
| Langues : |
Anglais (eng) |
| Catégories : |
[DIVERS] personne:famille:adolescent
[DIVERS] personne:par âge:jeune
[TABAC] étude:recherche:recherche clinique:essai clinique randomisé
[TABAC] sevrage tabagique
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
Objective. Well-documented challenges have hampered both intervention development and research in teen smoking cessation. Addressing these challenges, the Hutchinson Study of High School Smoking (HS Study), the largest group-randomized trial in adolescent smoking cessation to date, incorporates several design innovations to investigate the effect of a counselor-initiated, individually tailored telephone counseling smoking cessation intervention for older adolescents. This paper presents and discusses these innovative design features, and baseline findings on the resulting study population. Method. The trial used a population-based survey to proactively identify and recruit all high school juniors who had smoked in the past month - potentially expanding intervention reach to all smokers, even those who smoked less than daily and those not motivated to quit. For ethical and intervention reasons, some nonsmokers were enrolled in the intervention, also. Other important design features included the random allocation of schools into experimental conditions (intervention vs. no-intervention control) and a multi-wave design. Results and conclusion. The design innovations address problems and challenges identified in adolescent smoking cessation literature. The heterogeneous baseline characteristics of the study population, well-balanced between the two arms, have three significant implications: They (1) demonstrate the effectiveness of the trial's design features, (2) highlight |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=2696 |
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Exemplaires (1)
|
| TA 002091 | TA 3.2.2.1 LIU A | Article/Périodique | Bibliothèque FARES | Tabac | Consultation sur place
Exclu du prêt |
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| Titre : |
Chronic pod-mod e-cigarette aerosol exposure inducesa aortic dysfunction in hypercholesterolemic mice : role of oxidative stress and inflammation |
| Type de document : |
document électronique |
| Auteurs : |
Yasmeen F. Farra, Auteur ; Jacqueline Matz, Auteur ; Hannah Wilker, Auteur |
| Editeur : |
Cold Spring Harbor Laboratory |
| Année de publication : |
2024 |
| Collection : |
bioRxiv : the preprint server for biology, ISSN 2692-8205 |
| Importance : |
p. 1-38 |
| Présentation : |
ill., graph. |
| Langues : |
Anglais (eng) |
| Catégories : |
[TABAC] chimie du tabac:tabac fumé:cigarette:cigarette électronique
[TABAC] étude
[TABAC] tabagisme:pathologie
[TABAC] tabagisme:tabagisme actif
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
Objective Electronic (e-)cigarettes are the most used tobacco product amongst youth, and adult smokers favor e-cigarettes over approved cessations aids. Despite the lower perceived harm of vaping compared to smoking, inhalation of e-cigarette aerosol elicits cardiovascular responses that may lead to permanent injury when repeated over time. We thus aimed to infer the long-term outcomes of vaping on the function and structure of the aorta and shed light on the underlying cellular and molecular mechanisms.
Approach and Results We exposed female hypercholesterolemic mice to either pod-mod e-cigarette aerosol or room air daily for 24 weeks. Chronic inhalation of e-cigarette aerosol triggered accumulation of inflam-matory signals systemically and within aortic tissues, as well as T lymphocyte accrual in the aortic wall. Reduced eNOS expression and enhanced ROS production following eNOS uncoupling and NADPH oxi-dase activation curbed nitric oxide availability in the aorta of mice exposed to e-cigarette aerosol, impairing the endothelium-dependent vasodilatation that regulates blood flow distribution. Inhalation of e-cigarette aerosol thickened and stiffened aortic tissues via collagen deposition and remodeling, hindering the storage of elastic energy and limiting the cyclic distensibility that enables the aorta to function as a pressure reservoir. These effects combined contributed to raising systolic and pulse pressure above control levels.
Conclusions Chronic inhalation of aerosol from pod-mod e-cigarettes promotes oxidative stress, inflammation, and fibrosis within aortic tissues, significantly impairing passive and vasoactive aortic functions. This evidence provides new insights on the biological processes that increase the risk for adverse cardio-vascular events as a result of pod-mod e-cigarette vaping. |
| En ligne : |
https://doi.org/10.1101/2024.01.30.578110 |
| Format de la ressource électronique : |
Article en ligne |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=10540 |
|
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| Titre : |
Cigarette smoke enhances {beta}-defensin 2 expression in rat airways via nuclear factor-{kappa}B activation |
| Type de document : |
texte imprimé |
| Auteurs : |
L. Chen, Auteur ; P.P. Sun, Auteur ; T. Wang, Auteur ; X. Wang, Auteur |
| Editeur : |
European Respiratory Society (ERS) |
| Année de publication : |
2010 |
| Collection : |
European Respiratory Journal num. 36 |
| Importance : |
p.638-645 |
| Présentation : |
graph., ill. |
| Langues : |
Anglais (eng) |
| Catégories : |
[TABAC] chimie du tabac:fumée
[TABAC] étude
[TABAC] tabagisme:pathologie:pathologie respiratoire:broncho-pneumopathie chronique obstructive
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
β-defensin 2 (BD-2), an antimicrobial peptide, participates in airway defence. Cigarette smoke (CS) is a major risk factor for the development of chronic obstructive pulmonary disease. This study mainly aims to investigate the effect of CS on rat BD-2 (rBD-2) expression in rat airways. Rats were exposed to CS and treated with caffeic acid phenethyl ester (CAPE), a nuclear factor (NF)-κB inhibitor, or astragaloside IV (AS-IV), an active ingredient of Astragalus mongholicus. Besides the analysis of bronchoalveolar lavage fluid (BALF) and histological changes after CS exposure, rBD-2 expression was investigated with immunohistochemistry, reverse transcription PCR and ELISA. Total glutathione and nitric oxide (NO) levels in rat lungs were also detected. CS exposure markedly increased rBD-2 immunoreactivity, as well as rBD-2 mRNA and protein levels in rat airways, which were inhibited by CAPE treatment. Moreover, associated airway inflammation induced by CS was demonstrated by histological changes, increased cell counts and pro-inflammatory cytokines in BALF, and NF-κB activation and high levels of total glutathione and NO, which were all reversed by AS-IV in a dose-dependent fashion. In conclusion, CS exposure induces rBD-2 expression in rat airways via a NF-κB-dependent pathway, and AS-IV attenuates CS-induced airway inflammation due to its anti-inflammatory and antioxidant properties, at least partly through NF-κB inactivation. |
| En ligne : |
https://erj.ersjournals.com/content/36/3/638.long |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=8075 |
|
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| Titre : |
Comparison of conventional cigarette and electronic cigarettes on P53, PTEN, and VEGF genes expression in rat kidney |
| Type de document : |
document électronique |
| Auteurs : |
Fatemeh Jahanshahi, Auteur ; Zahra Tootian, Auteur ; Fateme Guitynavard, Auteur |
| Editeur : |
Tehran University of Medical Sciences |
| Année de publication : |
2023 |
| Collection : |
Translational Research in Urology, ISSN 2717-042X num. 5(4) |
| Importance : |
p. 167-174 |
| Présentation : |
ill., graph.; tab |
| Langues : |
Anglais (eng) |
| Catégories : |
[TABAC] chimie du tabac:fumée
[TABAC] chimie du tabac:tabac fumé:cigarette:cigarette électronique
[TABAC] étude
[TABAC] tabagisme:risque:facteur associé:génétique
|
| Mots-clés : |
rat, animal |
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
Introduction: Various genetic alterations and gene expression changes are involved in the pathogenesis of kidney tumours. The P53, PTEN, and VEGF gene expression pattern in rat kidney tissue exposed to conventional cigarettes and e-cigs was evaluated.
Method: In the study, 60 Wistar rats were categorized into three groups (10): no smoke, c-cig smoke, and e-cig smoke. Three cigarette butts used daily with the suction device were exposed for 40 minutes. After four months, kidney tissue was removed, tissue RNA was extracted, cDNA was made, and changes in mRNA expression of genes were evaluated by real-time PCR. Moreover, kidney biopsies were assessed for histopathological changes.
Result: E-cig smoke might contribute to kidney cancer more than C-cigarette through upregulating VEGF and downregulating PTEN and P53 (P-value >0.05). The effect of e-cigarettes in female rats was more significant than in male rats. The histopathological investigation revealed decreased glomerular space in e-cig male rats, increased tubular necrosis in e-cig, Interstitial infiltration in c-cig, and Hyperemia in c-cig in female rats were reported.
Conclusion: Since E-cigarette smoke displayed overexpressed VEGF and down-expressed PTEN and P53 expression than C-cigarette, it has a higher potential for kidney carcinogenesis. |
| En ligne : |
https://doi.org/10.22034/tru.2023.421288.1162 |
| Format de la ressource électronique : |
Article en ligne |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=10582 |
|
Aucun avis, veuillez vous identifier pour ajouter le vôtre !
| Titre : |
Factors influencing JUUL e-cigarette nicotine vapour-induced reward, withdrawal, pharmacokinetics and brain connectivity in rats : sex matters |
| Type de document : |
document électronique |
| Auteurs : |
Jude A. Frie, Auteur ; Patrick McCunn, Auteur ; Amr Eed, Auteur |
| Editeur : |
Springer Nature |
| Année de publication : |
2023 |
| Collection : |
Neuropsychopharmacology, ISSN 2948-1570 num. 49 |
| Importance : |
p. 782-795 |
| Présentation : |
ill., graph.; tab. |
| Langues : |
Anglais (eng) |
| Catégories : |
[TABAC] chimie du tabac:constituant:alcaloïde:nicotine
[TABAC] chimie du tabac:tabac fumé:cigarette:cigarette électronique
[TABAC] sevrage tabagique:méthode de sevrage:méthode individuelle:approche pharmacologique
[TABAC] tabagisme:aspect psychologique:comportement
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
Though vaping likely represents a safer alternative to smoking, it is not without risks, many of which are not well understood, especially for vulnerable populations. Here we evaluate the sex- and age-dependent effects of JUUL nicotine vapour in rats. Following passive nicotine vapour exposures (from 59 mg/ml JUUL nicotine pods), rats were evaluated for reward-like behaviour, locomotion, and precipitated withdrawal. Pharmacokinetics of nicotine and its metabolites in brain and plasma and the long-term impact of nicotine vapour exposure on functional magnetic resonance imaging-based brain connectivity were assessed. Adult female rats acquired conditioned place preference (CPP) at a high dose (600 s of exposure) of nicotine vapour while female adolescents, as well as male adults and adolescents did not. Adult and adolescent male rats displayed nicotine vapour-induced precipitated withdrawal and hyperlocomotion, while both adult and adolescent female rats did not. Adult females showed higher venous and arterial plasma and brain nicotine and nicotine metabolite concentrations compared to adult males and adolescent females. Adolescent females showed higher brain nicotine concentration compared to adolescent males. Both network-based statistics and between-component group connectivity analyses uncovered reduced connectivity in nicotine-exposed rats, with a significant group by sex interaction observed in both analyses. The short- and long-term effects of nicotine vapour are affected by sex and age, with distinct behavioural, pharmacokinetic, and altered network connectivity outcomes dependent on these variables. |
| En ligne : |
https://doi.org/10.1038/s41386-023-01773-3 |
| Format de la ressource électronique : |
Article en ligne |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=10590 |
|
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(2023)
| Titre : |
Fourteen-day e-cigarette eposure disrupts ventilation patterns and serum IL-1ꞵ levels in adolescent rats |
| Type de document : |
document électronique |
| Auteurs : |
Mila Fisher, Auteur ; Alicia, M Peters, Auteur ; Jennifer A. Stokes, Auteur |
| Editeur : |
Houston [Etats-Unis] : Journal of student research |
| Année de publication : |
2023 |
| Collection : |
Journal of student research num. 12 : 4 |
| Importance : |
12 p. |
| Présentation : |
graph,tab. |
| ISBN/ISSN/EAN : |
2167-1907 |
| Langues : |
Anglais (eng) |
| Catégories : |
[TABAC] CANDIDATS:e-cigarette
[TABAC] étude
[TABAC] tabagisme:pathologie:pathologie respiratoire
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
E-cigarettes are devices used to deliver vaporized liquids often containing nicotine and other chemicals. The purpose of this study was to investigate the effects of 14 days of e-cigarette vapor exposure in adolescent rats on lung function and inflammatory cytokine expression. Seventeen male Long-Evans rats were assigned to vape or air groups. The animals were exposed to either air or 5% nicotine vapor using a whole-body exposure chamber, once a day for ten minutes for fourteen consecutive days. Ventilation recordings were completed on day 0 (before exposure) and day 15 (after exposure) using unrestrained whole-body plethysmography. Both lung tissue and blood was collected for mo-lecular assays. Baseline and post-exposure ventilation data were compared between air and vape groups across three different parameters: frequency, tidal volume, and minute ventilation. These parameters were compared resulting in three distinct 2x2 (time x treatment) Mixed Model ANOVAs. Between baseline and post-treatment measurements there was a significant decrease in values in minute ventilation (p=0.0312) and tidal volume (p=0.0031). Between treatment groups there was a significant difference in minute ventilation (p=0.0128) and frequency (p=0042). There were also significant alterations to the presence of IL-1β in the serum (p=0.0003), but not lung tissue (p=0.6525). In conclusion, following two weeks of e-cigarette vapor exposure, ventilation patterns were altered in the vapor exposed animals which decreased in tidal volume and minute ventilation suggesting possible impairment of lung function and e-cigarette exposure modified the expression of pro-inflammatory cytokine, IL-1ꞵ. |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=10184 |
|  |
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(16/05/2013)
Exemplaires (1)
|
| TA 004767 | TA 3.2.2.1 BOU C | Article/Périodique | Bibliothèque FARES | Tabac | Consultation sur place
Exclu du prêt |
Aucun avis, veuillez vous identifier pour ajouter le vôtre !
| Titre : |
Mechanisms of clearance of nontypeable Haemophilus influenzae from cigarette smoke-exposed mouse lung |
| Type de document : |
texte imprimé |
| Auteurs : |
G.J. Gaschler, Auteur ; C.C.J. Zavitz, Auteur ; C.M.T. Bauer, Auteur ; M.R. Stämpfli, Auteur |
| Editeur : |
European Respiratory Society (ERS) |
| Année de publication : |
2010 |
| Collection : |
European Respiratory Journal num. 36 |
| Importance : |
p. 1131-1142 |
| Présentation : |
graph. |
| Langues : |
Anglais (eng) |
| Catégories : |
[TABAC] chimie du tabac:fumée
[TABAC] tabagisme:pathologie:pathologie respiratoire:broncho-pneumopathie chronique obstructive
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
Inflammation is prevalent in all stages of chronic obstructive pulmonary disease, and, furthermore, individuals undergo periods of exacerbation, during which pulmonary inflammation increases, often a result of bacterial infection. The present study investigates the in vivo consequences of cigarette smoke exposure on bacterial challenge with nontypeable Haemophilus influenzae (NTHi). BALB/c and C57 black 6 (C57BL/6) mice were exposed to cigarette smoke once or twice daily for a total period of 8 weeks. Exacerbated inflammation was observed in cigarette smoke-exposed compared to room-air-exposed mice following challenge with live or heat-inactivated NTHi. Accelerated clearance of live NTHi from cigarette smoke-exposed mice was independent of the establishment of chronic inflammation or direct toxic effects of cigarette smoke components on bacteria. Mechanistically, a cell-free factor in the bronchoalveolar lavage fluid contributed to accelerated clearance following passive transfer to naive mice. Further investigation demonstrated increased titres of immunoglobulin A in the bronchoalveolar lavage fluid, but not the blood, of cigarette smoke-exposed mice, including increased titres of NTHi-specific immunoglobulin A, whereas heavy chain joining element (J(H))(-/-) B-cell-deficient cigarette smoke-exposed mice did not demonstrate decreased bacterial burden following challenge. The present results demonstrate that cigarette smoke exposure results in exacerbated inflammation following challenge with NTHi, as well as increased titres of antibodies that contribute to bacterial clearance. |
| En ligne : |
https://erj.ersjournals.com/content/36/5/1131 |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=8074 |
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Exemplaires (1)
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| TA 005625 | TA 3.2.2.1 GAS M | Article/Périodique | Bibliothèque FARES | Tabac | Consultation sur place
Exclu du prêt |
Aucun avis, veuillez vous identifier pour ajouter le vôtre !
(2/09/2010)
| Titre : |
Polyarthrite rhumatoïde : la cessation tabagique est impérative |
| Type de document : |
texte imprimé |
| Auteurs : |
Jean-Luc Schouvelier, |
| Année de publication : |
2/09/2010 |
| Importance : |
p.26 |
| Langues : |
Français (fre) |
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
Coup sur coup, trois études présentées au cours du récent congrès de l'EULAR, qui se tenait à Rome, révèlent que le tabagisme actif constitue non seulement un facteur de risque de développement et de sévérité de polyarthrite rhumatoïde, mais aussi qu'il diminue significativement l'efficacité des biothérapies ciblées par anti-TNF alpha. |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=7693 |
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Exemplaires (1)
|
| TA 002646 | TA 3.2.2.2.1 SCH P | Article/Périodique | Bibliothèque FARES | Tabac | Consultation sur place
Exclu du prêt |
Aucun avis, veuillez vous identifier pour ajouter le vôtre !
| Titre : |
Pulmonary hypertension in smoking mice over-expressing protease-activated receptor-2 |
| Type de document : |
texte imprimé |
| Auteurs : |
G. De Cunto, Auteur ; S. Cardini, Auteur ; G. Cirino, Auteur ; G. Lungarella, Auteur |
| Editeur : |
European Respiratory Society (ERS) |
| Année de publication : |
2011 |
| Collection : |
European Respiratory Journal num. 37 |
| Importance : |
p.823-834 |
| Présentation : |
tab., graph., ill. |
| Langues : |
Anglais (eng) |
| Catégories : |
[TABAC] chimie du tabac:fumée
[TABAC] étude
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
The mechanism(s) involved in the development of pulmonary hypertension (PH) in COPD is still the object of investigation. Cigarette smoke (CS) may lead to remodelling of intrapulmonary vessels and dynamic changes in vascular function, at least in some smokers. A role for proteases in PH has been recently put forward. We investigated, in smoking mice, the role of protease-activated receptor (PAR)-2 in the pathogenesis of PH associated with emphysema. We demonstrated that CS exposure can modulate PAR-2 expression in mouse lung. Acute CS exposure induces in wildtype (WT) and in transgenic mice over-expressing PAR-2 (FVB(PAR-2-TgN)) a similar degree of neutrophil influx in bronchoalveolar lavage fluids. After chronic CS exposure WT and FVB(PAR-2-TgN) mice show emphysema, but only transgenic mice develop muscularisation of small intrapulmonary vessels that precedes the development of PH (~45% increase) and right ventricular hypertrophy. Smoking in FVB(PAR-2-TgN) mice results in an imbalance between vasoconstrictors (especially endothelin-1) and vasodilators (i.e. vascular endothelial growth factor, endothelial nitric oxide synthase and inducible nitric oxide synthase) and enhanced production of growth factors involved both in fibroblast-smooth muscle cell transaction (i.e. platelet-derived growth factor (PDGF) and transforming growth factor β) and vascular cell proliferation (PDGF). PAR-2 signalling can influence the production and release of many factors, which may play a role in the development of PH in smokers.
PMID: 20693251 DOI: 10.1183/09031936.00060210 |
| En ligne : |
https://erj.ersjournals.com/content/37/4/823.long |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=8072 |
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| Titre : |
A single 10-minute e-cigarette vapor exposure reduces tidal volume and minute ventilation in normoxia and normobaric hypoxia in adult rats |
| Type de document : |
document électronique |
| Auteurs : |
Jennifer A. Stokes, Auteur ; Mila Fisher, Auteur |
| Editeur : |
Springer Nature |
| Année de publication : |
2023 |
| Collection : |
Cureus, ISSN 2168-8184 num. 15(10) |
| Importance : |
8 p. |
| Présentation : |
ill. ; graph. |
| Langues : |
Anglais (eng) |
| Catégories : |
[DIVERS] anatomie:corps humain:appareil respiratoire:poumon
[TABAC] CANDIDATS:e-cigarette
[TABAC] chimie du tabac:tabac fumé:cigarette:cigarette électronique
[TABAC] tabagisme:pathologie:pathologie respiratoire
[TABAC] tabagisme:tabagisme passif
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
The present study investigated the effects of a single 10-minute exposure to e-cigarette vapor on ventilation in adult male Long-Evans rats. Ventilation was recorded using awake, unrestrained whole-body plethysmography. Baseline recordings were taken the day before full-body exposure to either room air (n = 9; air control group) or e-cigarette vapor (n = 9; treatment group). Post-exposure recordings were taken immediately after the 10-minute room air or vapor exposure. As part of the ventilation protocol, in addition to recording the subject’s ventilation in room air, the subjects were also exposed to 10% oxygen (balanced with nitrogen) to assess the effects of e-cigarette vapor on an increased drive to breathe. Ventilation data were analyzed using a 2x2x2 mixed-model ANOVA measuring treatment (vape vs. air) x time (baseline vs. post-treatment) x condition (normoxia vs. hypoxia) for breathing frequency, tidal volume, and minute ventilation. Breathing frequency increased in both treatment groups (air and vape) with exposure to normobaric hypoxia (p < 0.001), with no effect of time (baseline vs. post-treatment) for either group. Tidal volume increased in both treatment groups (air and vape) with exposure to normobaric hypoxia (p < 0.001), and an effect of time (baseline vs. post-treatment) was observed (p = 0.010) for the vape group. Minute ventilation increased in both treatment groups (air and vape) with exposure to normobaric hypoxia (p < 0.001), and an effect of time (baseline vs. post-treatment) was observed (p < 0.001) for the vape group.
In conclusion, immediately following a single 10-minute e-cigarette vapor exposure, both tidal volume and minute ventilation were reduced during normoxia and normobaric hypoxia, indicating a decrease in ventilation after a single 10-minute e-cigarette vapor exposure. Furthermore, this exposure also blunted the physiological response to acute hypoxia exposure. Subjects in the vape group, while breathing more rapidly as expected, experienced shallower breathing than the air group during hypoxia. The findings in this study confirm that vaping could result in reduced lung function. |
| En ligne : |
https://www.doi.org/10.7759/cureus.46735 |
| Format de la ressource électronique : |
Article en ligne |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=10713 |
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(7/05/2008)
Exemplaires (1)
|
| TA 001998 | TA 3.2.1 KEN S | Article/Périodique | Bibliothèque FARES | Tabac | Consultation sur place
Exclu du prêt |
Aucun avis, veuillez vous identifier pour ajouter le vôtre !
(2000)
| Titre : |
Tabagisme et débit expiratoire de pointe |
| Type de document : |
texte imprimé |
| Auteurs : |
B. Chini, Auteur |
| Editeur : |
Paris [France] : Elsevier |
| Année de publication : |
2000 |
| Collection : |
Archives des Maladies Professionnelles et l'environnement, ISSN 1775-8785 num. 61 (5) |
| Importance : |
p.304-309 |
| Catégories : |
[TABAC] étude
[TABAC] étude:enquête
[TABAC] tabagisme
|
| Index. décimale : |
TA 3.2.2.1 Expérimentation |
| Résumé : |
Dans une enquête portant sur 485 homms et 352 femmes,de 18 à 64 ans examinés lors des consultations de médecine du travail, l'effet du tabagisme sur le débit expiratoire de pointe (DEP) a été étudié en comparant les courbes de régression du DEP en fonction des paramètres connus pour influencer cette variable, tels que la taille, l'âge et le poids, pour les différents groupes : fumeurs, non-fumeurs, et ex-fumeurs. Cette étude conclut à un effet significatif du tabac sur le DEP, en mettant en évidence un déclin plus rapide du DEP en fonction de l'âge chez les hommes fumeurs comparés aux hommes non-fumeurs. L'effet du tabac n'apparaît pas chez les femmes, mais le faible effectif de femmes de plus de 35 ans ne permet pas de tirer cette conclusion définitive. |
| Permalink : |
https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=1459 |
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Exemplaires (1)
|
| TA 002273 | TA 3.2.2.1. CHI T | Article/Périodique | Bibliothèque FARES | Tabac | Consultation sur place
Exclu du prêt |
Aucun avis, veuillez vous identifier pour ajouter le vôtre !
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